What are the cellular targets of those metabolites? Could we develop some ligands to identify the enzyme responsible for arachidonic acid metabolism?
Tat-mediated protein transduction is a means to deliver recombinant proteins into cells and represents a novel therapeutic approach, but it is fraught with challenges in expression and purification of the recombinant fusion proteins.
What could we add to the buffer to keep the proteins from precipitating?
I may also talk about a hare-brained idea I would like to invite collaboration on, to develop a FRET parallel for NMR imaging.